José A. Martínez Orgado-rekin lankidetzan egindako argitalpenak (4)

2012

  1. β-Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR-α, PPAR-γ and TRPV1, but not CB1 or CB2 receptors

    British Journal of Pharmacology, Vol. 166, Núm. 4, pp. 1474-1489

2010

  1. The neuroprotective effect of cannabidiol in an in vitro model of newborn hypoxic-ischemic brain damage in mice is mediated by CB2 and adenosine receptors

    Neurobiology of Disease, Vol. 37, Núm. 2, pp. 434-440

2009

  1. The activation of cannabinoid CB2 receptors stimulates in situ and in vitro beta-amyloid removal by human macrophages

    Brain Research, Vol. 1283, pp. 148-154

2008

  1. Neuroprotective effects of the nonpsychoactive cannabinoid cannabidiol in hypoxic-ischemic newborn piglets

    Pediatric Research, Vol. 64, Núm. 6, pp. 653-658