Publikationen in Zusammenarbeit mit Forschern von Icahn School of Medicine at Mount Sinai (24)

2024

  1. Factors Associated With Coronary Angiography Performed Within 6 Months of Randomization to the Conservative Strategy in the ISCHEMIA Trial

    Circulation: Cardiovascular Interventions, Vol. 17, Núm. 6, pp. 457-473

  2. Global, regional, and national burden of stroke and its risk factors, 1990–2021: a systematic analysis for the Global Burden of Disease Study 2021

    The Lancet Neurology, Vol. 23, Núm. 10, pp. 973-1003

  3. β3-Adrenergic receptor overexpression in cardiomyocytes preconditions mitochondria to withstand ischemia–reperfusion injury

    Basic Research in Cardiology, Vol. 119, Núm. 5, pp. 773-794

2022

  1. Beta-3 adrenergic receptor overexpression reverses aortic stenosis–induced heart failure and restores balanced mitochondrial dynamics

    Basic Research in Cardiology, Vol. 117, Núm. 1

  2. Mapping age- and sex-specific HIV prevalence in adults in sub-Saharan Africa, 2000–2018

    BMC Medicine, Vol. 20, Núm. 1

2021

  1. Storage proteins are driving pediatric hazelnut allergy in a lipid transfer protein-rich area

    Foods, Vol. 10, Núm. 10

2018

  1. Mirabegron, a Clinically Approved β3 Adrenergic Receptor Agonist, Does Not Reduce Infarct Size in a Swine Model of Reperfused Myocardial Infarction

    Journal of Cardiovascular Translational Research, Vol. 11, Núm. 4, pp. 310-318

2017

  1. Neutrophil stunning by metoprolol reduces infarct size

    Nature Communications, Vol. 8

2016

  1. Advances in Exercise, Fitness, and Performance Genomics in 2015

    Medicine and Science in Sports and Exercise, Vol. 48, Núm. 10, pp. 1906-1916

  2. Beta-3 adrenergic agonists reduce pulmonary vascular resistance and improve right ventricular performance in a porcine model of chronic pulmonary hypertension

    Basic Research in Cardiology, Vol. 111, Núm. 4

  3. Synthetic Studies of Neoclerodane Diterpenes from Salvia divinorum: Identification of a Potent and Centrally Acting μ Opioid Analgesic with Reduced Abuse Liability

    Journal of Medicinal Chemistry, Vol. 59, Núm. 24, pp. 11027-11038

2015

  1. Exercise triggers ARVC phenotype in mice expressing a disease-causing mutated version of human plakophilin-2

    Journal of the American College of Cardiology, Vol. 65, Núm. 14, pp. 1438-1450

  2. Impact of left ventricular hypertrophy on troponin release during acute myocardial infarction: New insights from a comprehensive translational study

    Journal of the American Heart Association, Vol. 4, Núm. 1

  3. Induction of sustained hypercholesterolemia by single adeno-associated virus-mediated gene transfer of mutant hPCSK9

    Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 35, Núm. 1, pp. 50-59

  4. Prevalence, vascular distribution, and multiterritorial extent of subclinical atherosclerosis in a middle-aged cohort the PESA (Progression of Early Subclinical Atherosclerosis) study

    Circulation, Vol. 131, Núm. 24, pp. 2104-2113

2014

  1. Cyclin A2 induces cardiac regeneration after myocardial infarction through cytokinesis of adult cardiomyocytes

    Science Translational Medicine, Vol. 6, Núm. 224

  2. Intra-scar perfusion heterogeneity by cardiac magnetic resonance in a porcine model of non-reperfused myocardial infarction

    International Journal of Cardiology, Vol. 176, Núm. 3, pp. 1288-1289

  3. Long-term benefit of early pre-reperfusion metoprolol administration in patients with acute myocardial infarction: Results from the Metocard-CNIC trial (Effect of Metoprolol in Cardioprotection during an Acute Myocardial Infarction)

    Journal of the American College of Cardiology, Vol. 63, Núm. 22, pp. 2356-2362

  4. Response to letter regarding article, "effect of early metoprolol on infarct size in ST-segment-elevation myocardial infarction patients undergoing primary percutaneous coronary intervention: The effect of metoprolol in cardioprotection during an acute myocardial infarction (METOCARD-CNIC) trial"

    Circulation

  5. β3 adrenergic receptor selective stimulation during ischemia/reperfusion improves cardiac function in translational models through inhibition of mPTP opening in cardiomyocytes

    Basic Research in Cardiology, Vol. 109, Núm. 4